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Probe-induced sensing capacitance manifested as time shifts of action potentials and increased synaptic input thresholds for somatic action potential initiation with linear dependence on the membrane density of the probe.
Next, we tested the impact of SERT-phosphovimentin association on the plasma membrane density of SERT.
The size and the plasma membrane density of the clusters significantly depend on the type of Cavβ present.
The plasma membrane density of SERT in CHO-SERT cells stimulated with 100 μM 5HT appeared lower than in cells stimulated with 10 μM 5HT.
At high concentrations, 5HT stimulation reduced the plasma membrane density of SERT and resulted in a loss of uptake function in platelet system (30%) that was more severe than that in the heterologous system (5.7%).
In exploring the impact of phosphovimentin-SERT association on the plasma membrane density of SERT, the second half of the same biotinylated samples were subjected to immunoblot analysis with pS56, phosphovimentin-Ab (Fig. 7D).
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Membrane densities of FNR and trimeric PSI have been estimated to 2000 and 2550 per μm2, respectively.
Importantly we also observed mineralocorticoid receptors at postsynaptic membrane densities of excitatory synapses.
Cho is a metabolic marker of membrane density and integrity.
Choline is a metabolic marker of membrane density and integrity.
Because the diffusion behavior and the local membrane density were coupled, averaging of the local molecule velocities showed that certain membrane areas were favorable for molecular motion while others were in a static phase.
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