The ER membrane changes were quantified by their fluorescence intensity.
Morphology and membrane changes were examined through light-inverted microscope.
Colistin toxicity was not observed and membrane changes were never needed during the treatment period [ 2, 4, 5].
ROS generation and mitochondrial membrane changes were also key events in apoptosis and necrosis; however, ATP depletion emerged only in oncosis.
These membrane changes are discernible using light and electron microscopy.
The pathophysiological significance of basement membrane changes is also likely to impact on clearance of fluids, solutes and toxins along the perivascular drainage route 9, 49.
Thromboembolic complications, including deep venous thrombosis, ischaemic stroke, intra-cardiac thrombus, pulmonary embolism and membrane circuit clotting requiring membrane change, were collected from the medical record.
Meanwhile, RD cells were infected with EV71 and membrane change was measured using flow cytometry.
In contrast, minimal membrane property changes were observed upon AA1 binding for liposomes mimicking mammalian cell membranes, which consist of neutral lipids and cholesterol.
The mitochondrial membrane potential changes were analyzed by flow cytometry using a mitochondria-selective dye JC-1 (5,5′,6,6′-tetrachloro-1,1′,3,3′-tetraethylbenzimidazolcarbocyanine; Sigma-Aldrich Chemicals, Germany) (Rees et al. 1991; White and Reynolds 1996).
In addition to the current recorded via the two-electrode voltage-clamp technique (TECV) membrane capacitance changes were monitored [26].
