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The membrane capacitance of the cells was calculated from the integral of the uncompensated capacitive artefact.
The membrane capacitance of the cells was measured by the integral of the uncompensated capacitive artefact (filtered at 6 kHz) evoked by a hyperpolarizing voltage step from −80 to −90 mV when Ba2+ ions (1 mM) were present in the bath solution to block the K+ conductance.
The membrane capacitance of the cells was measured by the integral of the uncompensated capacitive artifact (filtered at 6 kHz) evoked by a 10-mV voltage step in the presence of extracellular BaCl2 (1 mM).
Chlorpromazine markedly decreased the membrane capacitance of megakaryocytes, indicating the decreased number of invaginated plasma membranes, which was not detected by the fluorescent imaging techniques.
Finally, the membrane capacitance of Müller cells was slightly increased in β2−/−/γ3−/− mice indicating that these cells have a larger membrane area (Fig. 4D) – a sign for cellular hypertrophy as typical for reactive glial cells [45].
Briefly, under conventional whole-cell mode in a standard K+ internal solution, frog parathyroid cells displayed a resting potential of about −30 mV, input resistance of 13 GΩ and membrane capacitance of 8 pF.
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It is known that mitoplasts typically have membrane capacitances of 0.5 1.2 pF and proton current ((I_{{{text{UCP}}1}})) densities of 60 110 pA/pF (Bertholet et al. 2017).
Phase angle reflects the relative contributions of fluid (resistance) and cellular membranes (capacitance) of the human body.
Here we show that layer 2/3 pyramidal neurons from human temporal cortex (HL2/3 PCs) have a specific membrane capacitance (Cm) of ~0.5 µF/cm2, half of the commonly accepted "universal" value (~1 µform2) for biological membranes.
This finding implies that the normalized net effects of activation (i.e., response net charge per unit of membrane capacitance per unit of time) of α7* nAChRs expressed in CA1 pyramidal neurons is ∼20-fold weaker than the net response of activation of functional α7* nAChRs expressed in CA1 interneurons.
The densities of expression and thus, the normalized net effect of activation (i.e., response net charge per unit of membrane capacitance per unit of time) of native functional α7* nAChRs in CA1 pyramidal neurons and CA1 interneurons were compared in whole-cell patch-clamp experiments in hippocampal slices.
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