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All inflammatory mediators were expressed at very low levels in the livers of RCD-fed hamsters.
Therefore, stimulated levels of mediators were expressed as fold change over constitutive levels.
Because we were aware of between-animal biological variation for primary cell cultures, for analysis of the impact of siRNA or inhibitors, the concentrations of inflammatory mediators were expressed for each animal as a percentage of the response to treatment with each PAMP.
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Cytokines A wide range of mediators are expressed and secreted as intercellular signals from activated MCs or other immune and non-immune cells during Yin and Yang processes.
Many proangiogenic mediators are expressed in RA, including vascular endothelial growth factor (VEGF).
The concentrations of inflammatory mediators are expressed as a percentage of the response to PAM or FSL-1 to examine the impact of the targeting siRNA.
Within the youth treatment outcome literature, the importance of identifying mediators was expressed more than a decade ago (e.g., Holmbeck 1997) and continues today (e.g., Kendall 2009).
We have recently demonstrated that VEGF-C and Flt-4, which are established mediators of lymphangiogenesis [ 55, 56], were expressed in the tumour cells of a large proportion (88%to95%5%) of DCIS specimens [ 29].
Data were expressed as the ratio between the inflammatory mediator CE and the GAPDH CE, yielding the relative expression (RE).
Demographic data were expressed as mean ± standard deviation (SD) and serum and joint fluid mediator levels as median (interquartile range, IQR).
While several inflammatory mediators were consistently expressed in all PLN from E-IAA positive and negative mice, few of these molecules were expressed in a significantly regulated manner (data not shown).
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