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Exact(10)
In a first step, pH profiles and catalytic constants of laccase for the redox mediators were determined.
In this study, BV-2 cells were stimulated with LPS in the presence or absence of C60 COOH pretreatment for 6 h, and then the levels of various pro-inflammatory mediators were determined.
Levels of inflammatory mediators were determined using enzyme-linked immunosorbent assay and between-group differences (IM and NIM; NS and FS) were evaluated using the Wilcoxon signed-rank test.
Myeloperoxidase (MPO) activity, transepithelial permeability and proinflammatory mediators were determined in whole colon or proximal and distal parts of colon.
Concentrations of all mediators were determined by ELISA as described in the Materials and methods section.
mRNA levels of inflammatory mediators were determined in villous tissue from normal term or RFM pregnancies.
Similar(50)
Protein expression of vascular mediators was determined.
Critical involvement of selected mediators was determined by using RNA interference and chemical inhibitors.
The origin of inflammatory mediators was determined using double IF colocalization studies.
The synovial expression of these and several T cell and DC/macrophage-related mediators was determined by qPCR (Table 1).
Mice (n = 15 in each group) were injected i.a. with 1 U/mouse of collagenase on Day 0 and Day 2. Synovial extract was collected separately from each animal at Day 7 of CIOA and the level of several mediators was determined by ELISA Studentt's t-test; *** P < 0.001 vs healthy mice.
More suggestions(15)
associates were determined
facilitators were determined
mediators were increased
mediators were entered
mediators were analyzed
mediators were eluted
mediators were examined
mediators were associated
mediators were related
mediators were surprised
mediators were correlated
mediators were quantified
mediators were evaluated
mediators were called
mediators were changed
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