Sentence examples for mediators of acute from inspiring English sources
T lymphocytes are known to be the principal mediators of acute allograft rejection, infiltrating graft tissues and interceding cell-mediated cytotoxicity reactions.
This approach to protein network analysis identified novel mediators of acute lung injury and protein pathways were redundant and involved in multiple biological processes [51].
The main objective of this study was to evaluate correlative relationships between rumen lipopolysaccharide (LPS) and mediators of acute phase response with milk fat yield and efficiency in dairy cows challenged with graded amounts of barley grain in the diet.
We hypothesized that injurious mediators in stored blood might potentiate acute lung injury in the setting of other mediators of acute lung injury, such as sepsis and that this potentiation would not be limited to a 6-hour window after transfusion.
Donor DCs, recipient DCs and T cells are some of the critical cellular mediators of acute allograft rejection as defects in these cells can significantly impair the ability of recipients to reject organ transplants [17] [19].
Prostaglandins are important mediators of acute and chronic inflammation [ 132].
NADPH oxidase 1 was reported to act as an important mediator of acute lung injury mediated by oxidative stress, particularly in alveolar cells.
Cyclooxygenase-2 (COX-2) is a mediator of acute and chronic inflammation, and drugs designed to specifically target this enzyme have achieved widespread clinical use.
NO is considered an important mediator of acute lung injury (ALI) and is endogenously produced by NO synthase 2 (NOS-2), an enzyme upregulated in both ARDS patients and animal ALI models [98 100].
Presently, MIF is recognized as a pleiotropic cytokine that functions as a pivotal mediator of acute and chronic inflammation and is synthesized by a variety of cell types and organs [3] [5].
In PRV-induced cystitis, both genetic and pharmacologic data presented here suggest that mast cells mediate cystitis pain via H1R and H2R, whereas genetic data exclude TNF as a significant mediator of acute mast cell-dependent pelvic pain.
