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Mediators evaluated by the mother at 4 months postpartum were used in the analysis.
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We did not identify a correlation between preoperative NRS scores and levels of the different inflammatory mediators evaluated.
The soluble mediator concentration was evaluated by incubating, normal cord blood CD34+ cells over ECFC layers, obtained from healthy donors and low-risk MDS patients.
The activation of cells and parasiticidal activity were evaluated by mediator quantification and direct counting of infected macrophages.
Potential intrapersonal, interpersonal and environmental mediators of PA and screen time were evaluated by a standardized questionnaire.
Soluble mediators were first dichotomized using median concentrations and evaluated by log-rank test in a Kaplan-Meier model.
This hypothesis was evaluated by comparing levels of specific inflammatory mediators, TNF-α, IL-6, aNGFNGF, in injured and uninjured meniscal tissue and related knee joint synovium.
Activation of HSCs and their response to the pro-fibrogenic cytokine TGF-β was evaluated by gene expression of activation markers and signal mediators using RT-qPCR.
Granulocytes obtained from MM patients had greater amount of arginase-1 (a key mediator of G-MDSC immunosuppression) than MGUS or healthy subjects evaluated by RT-PCR (Parrinello and Romano, manuscript in preparation).
The enzymatic degradation of the three dyes was evaluated by employing a commercial laccase and ABTS as mediator (see Additional file 1: Annex S1).
Cells were treated with T (10 μg/ml) plus L (1 μM) for 48 hrs and inhibition of the HER pathway was evaluated by measuring phosphorylated EGFR, HER2, HER3, and key signal transduction mediators, including AKT and p44/42 MAPK.
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