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The role of glucocorticoids in mediating these changes in energy metabolism is still unclear because stocking density treatment was not associated with changes in adrenal secretion of cortisol following ACTH stimulation.
Together, the findings suggest age-related changes in the efficiency of using control processes to facilitate successful retrieval while highlighting the role of onset latency in mediating these changes.
The intermediate steps of signal transduction pathways mediating these changes are unknown.
Finally, we examined the changes in chromatin structure at the vgQ reporter gene directed by Gro and we explored the role of histone deacetylation in mediating these changes.
The signal transduction pathways mediating these changes remain unknown.
Moreover, the factors mediating these changes at a molecular level are not known.
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Changes in voltage-gated channel expression and/or function could mediate these changes in excitably after LTP (intrinsic plasticity).
EP tube morphogenesis via cell or cord hollowing may mediate these changes allowing type II EP bridge lumen formation [23].
Little is known about the mechanisms that mediate these changes, however we observed elevated PO activity during these later stage of aggregation, and note that DpoA appears to act as a suppressor of cell speed during chemotaxis, suggesting that the PO/Mipp1 signal pathway could be involved with these developmental changes.
Whether the ER mediates these changes in female heart is not clear.
The end-products of COX-2 activity are prostaglandins and thromboxanes which may mediate these changes in cancer cell progression.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com