Sentence examples for mediating the toxic from inspiring English sources
Aryl hydrocarbon receptor (AhR) is a ligand activated transcription factor involved in xenobiotic metabolism and in mediating the toxic effects of dioxin-like compounds.
The pulmonary extracellular lining could play a significant role mediating the toxic action of inhaled agents as well as the removal of those agents from the lungs.
However, we could not exclude the possibility that other iodide forms, molecules, or pathways are also involved in mediating the toxic effects of excess iodide.
The arylhydrocarbon receptor (AhR), a ubiquitous basic Helix-Loop-Helix (bHLH) receptor expressed in various tissues in vertebrate species, is best known for its role in mediating the toxic actions of dioxin.
These results highlight the importance of ER stress in mediating the toxic effects of galactose, and suggest that the UPR plays a protective role that could be harnessed in novel treatments for galactosaemia.
Its depletion could induce accumulation of NAPQI, which might mediate the toxic effect observed with AAP treatment through the mechanisms described above.
AhR is a ligand-activated transcription factor that mediates the toxic actions of environmental contaminants such as 2,3,7,8-tetrachlorodibenzo-p-dioxin 2,3,7,8-tetrachlorodibenzo-p-dioxin 2,3,7,8-tetrachlorodibenzo-p-dioxin 2,3,7,8-tetrachlorodibenzo-p-dioxin
A growing body of evidence suggests that several pathophysiologic pathways may mediate the toxic effects of glucose fluctuations.
Given the effect of prolonged exposure of fatty acids, divergent studies in animals with disruption of GPR40 gave rise to a controversy about the potential role of GPR40 to mediate the "toxic" effects of fatty acids.
To investigate whether Ynd1 mutants mediating the E4orf4 toxic signal, and specifically the cytosolic mutant, have retained the ability to interact with E4orf4, coimmunoprecipitation experiments were carried out.
The specificity and efficacy of TFEB in mediating the clearance of toxic Tau species and rescuing neurodegeneration is remarkable.
