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can switch from a classically activated (inflammatory macrophage or T effector cell) to an alternatively activated (pro-tumorigenic macrophage or Treg) state and what mediates this change.
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Can the seeming timelessness and spacelessness that mediate this change also be called physical?
Furthermore, the model suggests that a conformational change is required for catalysis and that residues Lys99 and Asp281 may be instrumental in mediating this change.
We therefore hypothesise that suppression of inflammation was the key factor mediating this change in EMPs.
Two alcohols that act antagonistically mediate this change: farnesol (intermediate in the synthesis of sterols) and tyrosol.
The development of new disease-modifying anti-rheumatic drugs (DMARDs), the successful application of a combination of DMARDs, and, last but not the least, the availability of biologicals mediated this change in treatment strategy.
Alternatively, lifestyle factors that were unaccounted for in this study may mediate the change in triglyceride and/or the associated modification of diabetes risk.
Short-term exposure to fine particulate matter (PM2.5) air pollution has been associated with altered DNA methylation in observational studies, but it remains unclear whether this change mediates the effects on cardiovascular biomarkers.
This change was mediated by particles known as X-bosons.
This change was mediated by a tissue-specific transcription mechanism.
It has been proposed that this change is mediated by the insect stress neurohormone octopamine.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com