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AMPA- and NMDA-receptor mediated currents were measured from recordings made at −60 and +40 mV, respectively (−70 and +40 mV in offspring of control and LPS-injected mice).

Inhibiting effects of menthol on nAChR mediated currents were observed for concentrations ≥2 μM, and maximum inhibition of 91% was observed at 500 μM.

AMPA-receptor mediated currents are important for transmitting inspiratory drive to XII MNs [14], [15].

Therefore 2-APB-dependent inhibition of STIM1 and Orai1 mediated currents are expected to result at least in part from a reduction of STIM1 punctae formation.

NMDA-mediated currents were measured at their peak amplitude and data expressed as mean ± s.e.m.m

KAR-mediated currents were also analysed in LII mEC stellate neurons.

The membrane potential was clamped at −10 mV, therefore GABAAR-mediated currents were detected as outward shifts.

Effects on TRPC5 channel-mediated currents were apparent at >1 µM pregnenolone sulphate.

AMPAR-mediated currents were decreased by the application of anti-kinesin and anti-dynein antibodies [ 43].

Kv1.3-mediated currents were elicited by 400 ms depolarizing pulses to +50 mV, from a holding potential of -60 mV.

However, the apparent desensitization time courses observed in α1β2γ2 and α3β2γ2 receptor-mediated currents were remarkably different.