Sentence examples for mediated by catecholamines from inspiring English sources

Perhaps another reason for the feeding paradigm-specific induction of Nocturnin is the fact that lipolysis in response to an acute stress, such as that presented to the restricted fed mice, is robust, transitory, and predominantly mediated by catecholamines [32].

Immune cell apoptosis is at least partly mediated by catecholamines, via α-adrenergic and β-adrenergic pathways.

This hypermetabolism is, at least in part, mediated by catecholamines and correlates with the severity of injury [ 14].

The effects of rolipram have also been suggested to be mediated by catecholamines [ 175] or by the stimulation of the adrenal and HPA axis [ 176, 177].

Bone marrow production and differentiation of monocytes is influenced by the adrenergic system [ 4, 5] and immune cell apoptosis is at least partly mediated by catecholamines, via alpha-adrenergic and beta-adrenergic pathways.

As such, the affinities of a range of β-adrenoceptor antagonists have been reported to be ≥1 order of magnitude lower when inhibiting responses mediated by CGP 12177 at the β1-adrenoceptor compared with those mediated by catecholamines in both animal and human tissue preparations (10, 14, 18).

The absence of increase in E/Ea and E/FPV does not exclude cardiac injury mediated by catecholamine release.

Available in vitro studies performed on AT biopsies, indicate that the difference in lipolysis mediated by catecholamine between upper body and lower body fat depots are more pronounced in women than in men (Richelsen, 1986).

This suggests that monoamines inhibit the activation of the HPA axis in these animals and indicates that acute stress-induced lymphocytopenia is, at least partially, mediated by catecholamine release.

The hypermetabolic response is in part mediated by endogenous catecholamines: shortly after severe burn or trauma, plasma catecholamine levels can increase up to 10-fold [ 92]. β-blockade following severe burns can reduce supraphysiologic thermogenesis, tachycardia, cardiac work and resting energy expenditure.

But it does not make explicit how a decreased neuronal energy supply mediated by hypofunctioning catecholamines might alter either cognitive performance based on frontal lobe function or account for ADHD symptomatology in general.