To determine their physics mechanism, we simulated the effect of Ag nanoparticles on infrared absorption by using a three-dimensional finite-difference time-domain (FDTD) method.
To test this mechanism, we simulated recycling by forcing GST-VCA to remain bound after nucleation and introducing a single binding reaction whereby cortactin returns the branch junction-bound GST-VCA to the pool of non-sequestered GST-VCA.
To investigate cellular mechanisms underlying jitter, we simulated SV release in single PF-MLI varicosities.
We simulated six mechanisms producing heterogeneity in susceptibility: (1) susceptibility was allocated at random to persons (i.e. no additional mechanism), (2) a household factor, (3, 4) a genetic factor (dominant or recessive), or (5, 6) half a household factor and half genetic.
To explore the dynamics of mariner transposition, we simulated the S-NEC mechanism of transpososome assembly in a second computer model ('Materials and methods').
We simulate cellular mechanisms including cell adhesion, growth, and division and diffusion of morphogens, to show that differential adhesion between the cells, diffusion of growth factors through the extracellular matrix, and the elastic properties of the apical ectodermal ridge together can produce the proper shape of the limb bud.
We simulate these mechanisms on a two dimensional square lattice with lattice spacing Δ.
Attracted by the excellent performance of neural visual systems, we simulate the mechanism of hypercolumns in the mammalian cortical area V1 that selectively responds to oriented bar stimuli.
Finally, we simulate and study the mechanism under various network structures and processor parameters.
Using our model for the zipper mechanism, we have successfully simulated phagocytic engulfment in a broad range of parameter values (see Figure 2, and movie Additional file 2). Figure 2A shows two different characteristic cup shapes we obtained.
