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In order to link circadian phenotypic variation with molecular mechanisms, we explored the genetic variation of well-characterized N. crassa clock genes across the 143 accessions.

To gain further insights into the possible mechanisms, we explored global response of iron deprived C. albicans cells through genome based microarray analyses.

To get insights into the underlying mechanisms, we explored whether AZA modulates the viability of NK cells.

As in conditions favoring proliferation FasL induces ERK1/2 activation, and BM MSCs maintain a stem cell phenotype through ERK1/2-dependent mechanisms, we explored ERK1/2 activation in BM MSCs treated with 0.5 ng/ml FasL.

To assess how expansion thoracoplasty affects biologic mechanisms, we explored (1) alveolar microstructure development (proliferating and macrophage cell fractions); and (2) capillary development and perfusion (vascular endothelial growth factor receptor 2 [VEGFR-2] immunohistochemistry and red blood cells in eosin-stained sections).

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Although we cannot clarify the underlying mechanisms, we explore two viewpoints.

The novel mechanisms we explore in this paper are based on provision-point contracts (also known as assurance contracts).

For each of these mechanisms, we explore examples of observational and experimental studies that have addressed the role of changing snow conditions on food web processes.

To confirm that ATRA promotes ERK activation by transcription-independent mechanism, we explored the effect of the pharmacologic inhibition of RARs.

The idea of these reduced forms is that, if the mechanism we explore is valid, we should expect that, ceteris paribus, being a temporary worker has a negative effect on the scores mainly through the reduction of the amount of OJT they undertake, and not so much through other alternative channels.

But it is possible that some RBf2 binding is directed by E2F-independent mechanisms, which we explore below.

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