These mechanisms may include feedback inhibition, forward feed, metabolite channelling, and counteractive metabolic and cellular perturbations.
Possible defect mechanisms may include transport through the interlayer of the layer-by-layer structure, leakage through gaps between the block copolymer and the channels, diffusion through the block copolymer matrix itself, and leakage through uncovered substrate pores.
These mechanisms may include parole, voluntary departure, stay of final order, section 107(c)(3 -based deferred action, or any other authorized form of continued presence, including applicable nonimmigrant visas.
Potential mechanisms may include carotid artery disease or cardiac valvular disease.
These mechanisms may include signalling between neurons and glia (satellite cells), both of which may express CGRP receptors (Fig. 2a).
Other possible mechanisms may include polarization of molten salt ions [35], multicomponent diffusional mechanisms of molten salts [36], or a combination effect of both.
These mechanisms may include, for example, revival of BBD in surviving corals from the previous year.
Possible mechanisms may include prevention of nutrient acquisition through alterations to inclusion properties.
Mechanisms may include SRA-induced suppression of p21Cip1 and p27Kip1 expression, and increased phosphorylation of Cdk1/Cdc2 (Figure 7).
As shown in an animal model, other possible mechanisms may include altered neuron excitability and neurotransmission [31].
Other steatogenic mechanisms may include enhancement of lipogenesis and/or inhibition of fatty acid β-oxidation in hepatocytes, following direct activation of Cnr2.
