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Wound healing is a complex cellular process involving various mechanisms making it intricate for designing a scaffold for therapeutic use.
It is also confirmed by the present work that the frequency response is very sensitive to the nature of the transfer mechanisms, making it theoretically feasible to discriminate between surface barrier control and intraparticle diffusion.
The advantage is that app developers do not need to write instructions for their products to work out how to interpret the different input mechanisms, making it easier for them to create new programs.
The regulation of epidermal homeostasis involves a complex interplay between different generic and genetic mechanisms, making it difficult for biologists to investigate except by focusing on separate discrete aspects.
To protect against infection, D. melanogaster employs a suite of cellular and humoral defense mechanisms, making it a good reference organism in which to dissect host responses (reviewed in [1], [2], [3], [4], [5], [6]).
First, the low establishment rates of small populations can be the result of any one of the above mechanisms, making it difficult to determine which is acting in any given case.
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Knowledge of its different dimensions, characteristics, and trigger mechanisms makes it possible to grasp the complexity and understand the consequences for the sufferer.
These kind of structural mechanisms make it much easier to deal with a vast pool of articles.
The variety of matching mechanisms makes it difficult to select a proper matching mechanism for a certain context.
The electromechanical interaction inside drive mechanisms makes it quite complicated to directly analyze the dynamic characteristics of an STFS.
Many commercial processes can take place by alternative reaction paths, and knowledge of the mechanisms makes it possible to choose reaction conditions that favour one path over others.
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