Haplotype 112 would be linked to a Y-chromosomal SR distorter, resulting in high population sex ratios during wet periods, and haplotype 557 to a Y-chromosomal SR suppressor, causing this haplotype to increase its frequency during dry periods when population sex ratios are low (schematic overview of the proposed mechanism in Table 2).
Further, in this work we have not examined the connection between the key support mechanisms presented in Table 1 or the relationships' information presented in Table 2 and the characteristics taxonomy presented in Fig. 1.
Using the mechanisms summarized in Table 1, the population of PC-IN pairs learns to increase PC output at unpaired PF signals (without CF signals) and decrease PC output at paired PF signals (conjunctive PF-CF activation) via the short-time scale component (LTP/LTD).
The key aspects of this model are qualitatively described in this section, with important equations and mechanisms tabulated in Table S3.
In the proposed mechanism, shown in Table 3, the operations at QAP can be divided into two major parts, TXOP duration calculation part (line 6-11) and checking for resource availability part (line 13-17).
This provides a single set of rate constants for both steps of the mechanism, shown in Table S5.
Anaemia mechanisms: The criteria used to define the three severe anaemia mechanisms are given in table 1. Etiological factors: Important etiological factors in the study population were defined by multivariate analysis and included: malaria, HIV, bacteraemia, hookworm infection and iron, vitamin B12 and vitamin A deficiency [10].
The proposed mechanisms are listed in Table 1 along with intervention types in which each mechanism is anticipated to operate.
Key mechanisms are summarized in Table 1.
The differences between the two different procurement mechanisms are defined in Table 1.
