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DOI: http://dx.doi.org/10.7554/eLife.04876.005 Two possible mechanisms can regulate Ca2+ release from ER upon mechanical laser-tweezer-traction.
Both mechanisms can regulate rRNA expression.
Basic sensorimotor mechanisms can regulate the activity of motoneurones (MNs) through different spinal pathways at segmental and intersegmental spinal cord levels.
Different cellular mechanisms can regulate the levels of transcripts carrying PTC.
These observations suggest that signaling mechanisms can regulate various vasoactive molecules and vice-versa.
Overall, this evidence supports the knowledge that miRNA-dependent epigenetic mechanisms can regulate the pathogenesis of human diseases in a highly integrative manner.
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While most trans-acting ncRNAs possess well-defined mechanisms that can regulate transcription or translation, they generally lack the ability to directly sense cellular signals.
There are also a variety of different mechanisms that can regulate each point of control.
The effects of LXR ligands on cancer cells may also involve non-genomic mechanisms, which can regulate cell proliferation and gene expression via post-translational modifications and signal transduction pathways.
To probe this idea further and, more generally, to examine the neural mechanisms that can regulate motor variability, we constructed a simple computational model of the HVC-RA-LMAN network.
Tight regulation mediated by sRNAs is often the result of interconnected circuits acting in feedforward or feedback mechanisms that can regulate transcriptional factors and two-component systems constituting nodal controls of complex biological networks.
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