Complex, inbred rodent models of autoimmunity provide an opportunity to dissect mechanisms and explore therapies in what is essentially a single donor recipient combination.
We investigate the current loss mechanisms and explore the potential for further improvement.
The present research was designed to examine the relative strength of both mechanisms and explore their boundary conditions.
Typical connectionist models, by contrast, follow a bottom-up approach, beginning with a characterization of neural mechanisms and exploring what macro-level functional phenomena might emerge.
These remote influences suggest the potential for skillful seasonal forecast exists, and we pro- pose to study these teleconnections, understand their mechanisms, and explore this potential for seasonal forecast of Mediterranean climate.
By following the biological perception mechanisms and exploring the saliency support region, the proposed method can achieve good performance to predict most of the fixations in the images that contain complex scenes and semantic objects.
Further investigations are needed to elucidate their precise generation mechanisms and explore their clinical significance in disease development and progression in a larger study population.
Further work is warranted in order to elucidate such mechanisms and explore the potential role of other circadian clock genes in the pathogenesis of T2DM.
Despite these clear challenges, the dire need for novel therapeutic paradigms highlights the importance of seeking out commonalities in HGT mechanisms and exploring the plausibility of targeting these pathways.
We harnessed the intron alternative splicing mechanism and explored three unique gene configurations that allow us to encode genetic structural variations into metabolic function.
