Suggested mechanisms range from upwelling of warm deep waters onto the continental shelf in response to variations in the westerly winds, to an influence of El Niño Southern Oscillation on sea surface temperatures.
These mechanisms range from the simple (substrate availability, thermodynamic properties of reactions, feedback inhibition, and organelle localization) to the elaborate (formation of multienzyme complexes and thiol-based redox switches).
The proposed coordinating mechanisms range from a network-based modeling approach to an assessment procedure for evaluating product architectures to the integration of relevant suppliers into the new product development (NPD) process to a goal programming modeling approach.
Underlying mechanisms range from structural changes of the light-sensitive cells over differences in conductance of photoreceptor membranes, or in synaptic signal transduction, up to altered levels of post- to presynaptic feedback modulation.
Quantity-based control mechanisms range from simple well permits to exchangeable pumping entitlements.
Hypothesized mechanisms range from peristaltic pumping to temperature sensing and response to fluid flow variations (rheotaxis), but little is known quantitatively about them.
Mechanisms range from the relatively simple, as with the plague bacillus, Yersinia pestis, to the elaborately intricate, as with parasites in the genera Plasmodium and Leishmania.
These suppressor mechanisms range from CD4+ regulatory T cells, functionally impaired dendritic cells, neutrophils, and monocytes to myeloid derived suppressor cells.
A combination of multiple oculomotor mechanisms (range error, global effect, or Bayesian estimation of target positions) working in parallel is also possible.
