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In this study, a novel three-dimensional approach is utilized for modeling the urine concentrating mechanism in rat renal outer medulla which is essentially focused on demonstrating the significance of tubule's architecture revealed in anatomic studies and physiological literature.
We studied this mechanism in rat right ventricles submitted to pulmonary arterial hypertension, because it is known that this ventricle is very vulnerable to the deleterious effects of pressure overload.
We therefore selected these herbs for the present study to test their neuroprotective ability and the associated mechanism in rat hippocampal slices subjected to oxygen-glucose deprivation (OGD).
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The present study was designed to investigate the potential therapeutic effect of trikatu, an herbal compound and its underlying molecular mechanism in rats with adjuvant-induced arthritis (AIA).
Our study was designed to investigate the antifibrotic effects of using short interference RNA (siRNA) to target TGF-β1 TGF-β1atin fibrosis and its mechepaticin rats exposed to a high-fibrosis and carbon tetrachlorits (CCL4).
However, in the NTS neurones, NO was shown to have a facilitatory role on the baroreflex mechanism in rats (Dias et al. 2005).
The aforementioned doses of IGF1 and IGFBP1 have previously been shown to be optimum in promoting the skin tissue repair mechanism in rats (Jyung et al., 1994).
Although the impact of endogenous NO on AT1 receptor-mediated control of RSNA has been explored (Kumagai et al. 1993, Eshima et al. 2000), there is less known regarding the contribution of centrally generated NO on AT2 receptor activation in the baroreflex regulatory mechanism in rats.
To understand the factors involved in this increase, we examined the roles of two transcription factors and epigenetic mechanisms in rat GCs.
However, apigenin did not affect the 0.1 μM phorbol 12,13-dibutyrate-induced 12,13-dibutyrate-induced 12,13-dibutyrate-inducedration range that relaxed the phenylephrine-contractionarterIC50=34.6±1.2ng that apigenin did not influence protein kinase C-mediated contractile mechanisms in rat aorta.
The present study aimed to examine the effect of diet-induced obesity on EDH-mediated function and the underlying mechanisms in rat mesenteric artery with myogenic tone.
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