All of them received mechanical ventilation (p = 0.1) and catecholamine infusion (p = 0.001).
In multivariate analysis, performance status >2 (p < 0.001) and mechanical ventilation (p = 0.039) were associated with worse in-hospital mortality.
Increased % of IE belonging to events, but not IE index was associated with increased ICU stay and duration of mechanical ventilation (p < 0.05, Kendall's tau).
Univariate analysis identified three factors associated with 1 year fatal outcome invasive mechanical ventilation (p = 0.0015), WHO performance status (p = 0.0001) and decline in functional status (p = 0.000).
In multivariate analysis, predictive factors for developing early high blood pressure were past history of ischemic heart disorder (p = 0.001) and support by mechanical ventilation (p = 0.01).
In univariate analysis, factors associated with 90-day mortality were: SAPS II (p = 0.0011), invasive mechanical ventilation (p < 0.0001), vasopressors (p = 0.0012) and renal replacement therapy (p = 0.045).
They displayed a significantly longer time under mechanical ventilation (P = 0.002) and ICU length of stay (P = 0.007) than patients with no therapeutic limitation.
The increased comorbidity (Charlson comorbidity index) and the ICU complications (pneumonia, sepsis-MODS) have significantly increased the duration of mechanical ventilation (p < 0.01).
Those patients showing lower levels were younger (p < 0.001) with lower APACHE-II (p < 0.001), did not need mechanical ventilation (p = 0.020) and were hemodinamically stable (p < 0.001).
Duration of mechanical ventilation (P = 0.01), intensive care unit stay (P = 0.01), and hospital stay (P = 0.0007) was significantly lower in the DN group.
Both mechanical ventilation (P =.02) and prolonged administration (>72 hours) of intravenous terbutaline (P =.02) were significantly associated with elevations in cTnT.
