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Sharp, atomically flat surface can be seen at the GaN template after the mechanical release of the epitaxial layers.
We selected endoscopy for observation and treatment because it has several merits: endoscopy ensures (1) viability of the bowel, shows the exact location of the damage and provides information regarding the mucosal appearance; (2) allows mechanical release of bowel obstruction and decompression of the dilated proximal bowel; and (3) is less invasive and more convenient than surgery [10].
As seen in Table 1, mechanical release of the collagen from contact with the edge of the plastic dish had minimal effect on tube formation.
Browning was used to measure the potential of phenolic oxidation after mechanical release of PPO proteins from their storage site in plastids [ 32].
By contrast, Fn levels in disease FPCLs (R0, Dis/ctrl ratio = 2.7) steadily decreased upon mechanical release of the matrices, while Fn levels in control lattices showed a slight increase during contraction (Fig. 6).
Similar(55)
It consists of the fabrication of vertical nanosheets self-standing on a silicon template, and their embedding in polydimethylsiloxane (PDMS) by a mechanical release during the peel-off of the cured polymer.
This paper offers a review of several prominent mechanical release methods for residual stress measurement and recent developments, focusing on the hole-drilling method combined with advanced optical sensing.
Moreover, the space occupied by the transplanted cells will press the surrounding host tissues, which may trigger by mechanical deformation further release of ATP from astrocytes.
Therefore, these data suggest that mechanical ventilation causes release of endogenous ligand(s) recognized by one or more MyD88-dependent receptors.
One possibility is that mechanical ventilation causes release of one or more endogenous ligands (i.e. signaling molecules generated by the host such as secreted cytokines or products resulting from tissue injury) for transmembrane receptors, associated with pro-inflammatory signaling.
This is consistent with the concept that the damage to, or stimulation of airway epithelium by various mediators including pollutants, allergens, viruses and even mechanical force stimulates release of inflammatory and pro-fibrotic mediators which drive and sustain asthmatic inflammation and airway remodelling (Fig. 3).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com