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Two methods of measuring endothelial dysfunction are flow-mediated dilatation (FMD) and peripheral arterial tonometry (PAT).
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Brachial FMD has been used in research settings for decades to measure endothelial dysfunction.
In our study patients with OSA had reduced FMD, a widely used non invasive method to measure endothelial dysfunction.
CD34+-cell levels tended to correlate negatively with all levels of measured endothelial dysfunction parameters at baseline.
OBJECTIVE: The purpose of this study was to assess endothelial dysfunction in women with preeclampsia by measuring endothelial microparticles.
In contrast to the PAT-derived measures of endothelial dysfunction, the validity and clinical significance of PAT-derived measures of arterial stiffness have yet to be determined.
We examined whether pregnancy complications are associated with post-pregnancy measures of endothelial dysfunction and arterial stiffness.
Increased urinary albumin creatinine ratio (ACR) has been associated with impaired endothelial-dependent vasodilation [8], a surrogate measure of endothelial dysfunction [9].
Further, the study did not include an alternative measure of endothelial dysfunction, such as peripheral artery tonometry.
Additionally, because biomarkers may be imprecise measures of endothelial dysfunction, we cannot categorically assert that the association of MetS with subclinical atherosclerosis is independent of endothelial dysfunction.
Women with SLE who did not have manifest CVD did not differ from age-matched controls with respect to FMD, a measure of endothelial dysfunction.
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