Most of these studies on growth laws measured bacteria in a steady-state situation, using a chemostat or balanced growth in a batch culture inoculated at high dilution [ 6, 9, 10, 14].
Again the statement of the spatial and regional context of measuring bacteria and inflammatory mediators applies here; in grinding up tissues you are getting the average of the entire tissue sample, while the most important measure is likely to be right at the site of the necrosis and leak.
Of course this interpretation should be considered with caution as we measured bacteria multiplication in vitro, and not in the insect host.
Assessing whales' health, however, isn't easy: Scientists hoping to measure bacteria and fungi in a whale's "breath"—the moist air it shoots from its blowhole need to get close enough to take a sample.
We and others have measured bacteria and viruses in the environment around poultry and swine houses.
The R2A agar is then considered as the gold standard for measuring heterotrophic bacteria in water [48].
The study did not measure gut bacteria in the participants to prove it was different, and other factors may have contributed to the link.
These predictions are supported both by the genetic data presented by the MFA studies [ 61, 62], which show an increase in edd and eda expression at high glucose concentrations, and MFA experimental results by Harcombe et al. [ 71], where fluxes through the ED pathway were measured in bacteria growing at growth rates above 1 h-1.
The ability of bacteria to degrade different concentrations of phenol was measured using culturing bacteria in different concentrations of phenol from 0.2 to 0.9 g/L.
In modeling the kinetics of receptor modification, we follow previous work that successfully describes the adaptive response measured in populations in bacteria (Shimizuet al., 2010; Pontius et al., 2013).
