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The assessment and analysis of trabecular microarchitecture, in combination with BMD measures, may thus provide substantial advancement in the field of osteoporosis diagnosis and therapy monitoring.
The correlations of TTO UVs with ophthalmic and functionality measures may thus be mainly driven by their drop among patients with end-stage POAG.
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The aMT, by contrast, is measured during contraction when such synapses are at or near firing potential: this measure may thus be less susceptible to tonic changes in these influences (Talelli et al. 2006).
Exclusion of marginalized groups through measurement of SES with ecological measures alone may thus lead to an underestimation and/or under-adjustment of SES effects.
Compared to the study reported here the time-scales involved in these experiments were much shorter (that is, seconds rather than minutes) and behavioural measures may be thus more closely related to instantaneous signal measures rather than monofractal dynamics that summarize signal complexity over an extended period of time.
The associated risk measures may be thus underestimated.
In particular, when parameters are difficult to be experimentally measured and may thus be affected by large variations, our methodology allows to study model behavior as parameters are varied with statistical techniques.
Quantitative measures of osteoporosis may thus be a more sensitive tool for assessment of inflammatory bone involvement in RA.
Its proximity to the usage measures according to PC1 may thus indicate that the usage measures are equally rapid indicators, if not more so.
Measuring a single pollutant may thus not serve as an adequate surrogate for other pollutants.
Depletions of these genes in a given wine strain as measured by array karyotyping may thus reflect a true deletion of that gene relative to the other wine strains, rather than just a decrease in copy number.
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