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However, given the rates the initial coarsening processes occur and the instrumental limitations described above, these measurements cannot determine the size at which this switch occurs.
However, qualitative measurements cannot determine substandard drugs.
Traditional bulk culture-based biochemical measurements cannot determine the variation of second messenger levels within populations.
Without a model, flux measurements cannot determine the components relative roles in enhancing the CO2 concentration in the carboxysome.
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Furthermore, as JSN is not a direct measure of cartilage damage, surrounding tissues may also be damaged, and the JSN measurement technique cannot determine the relative contributions of these tissues.
Without more fine-grained fMRI measurements, however, we cannot determine if the overlapping clusters between groups reflect activation of the same neuronal populations.
From the FRET measurements alone, we cannot determine potential binding interfaces of the compound to SERCA, but we speculate that these compounds do not bind to the SERCA2a-PLB interface, but rather affect FRET through allosteric changes in SERCA itself.
As with other bulk RNA sequence measurement, our methods cannot determine the absolute number of each Ig transcript in a given B cell population.
However, since the latent variables seem to have a different meaning in the two groups (lack of group measurement invariance), one cannot determine whether the difference between lagged coefficients is statistically significant.
But such measurements cannot define value.
Another is that it is change which is discontinuous and yet causal: one can make things to happen with measurement, even though one cannot determine the exact outcome.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com