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However, unlike the study reported by Tarrand et al. where serum levels or mRNA were measured, we evaluated LMP-1 expression by IHC directly on bone marrow specimens.
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Using linear (time and frequency domain) and nonlinear (approximate entropy and parameters of Poincare plots) measures, we evaluated the dynamic time course of the HRV response to the asphyxia insult and the effect of preconditioning on the autonomic neurocardiac control.
Using these measures, we evaluate both the quality of the simulated dialogs and the improvement of the new dialog strategy that is obtained with the interaction of the two modules.
For measuring sensitivity, we evaluated predicted genes with all lengths against annotated genes of certain length categories (up to 40 aa length, 41 to 80, 81 to 120, 121 to 160, 161 to 200).
We first examined how participants responded to each of these measures, then we evaluated the intercorrelations between the measures.
The 13 variables did not include biochemical measures, but we evaluated whether risk scores generated from the model were associated with blood serum measures obtained from a small SOF cohort during the baseline visit (n ≤ 400 for each of 37 measures considered).
"We have a number of measures that we evaluated that support this".
However, here we report results that include three SES measures that we evaluated for independent effects.
Using a repeated measures design we evaluated changes in pain and function over a 6-month period as compared to baseline.
To validate our measures further, we evaluated content validity, construct validity (i.e., convergent and discriminant validity) and composite scale reliability.
Since not all the LC-PUFAs were measured, we could not evaluate the same ratios in the n-3 pathway.
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