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As a complementary measure, we quantified the GC content in different coding and non-coding transcripts and ORFs.
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Of the PUFA variables measured, we quantified strong associations between FFQ intake estimates and erythrocyte membrane content of the two key long-chain omega-3 PUFAs, DHA and EPA.
Using these measures, we quantify the accuracy of derivatives and differences in terms of the noise level of the function.
To measure peptidoglycan, we quantified the component N-acetyl muramic acid (2-acetamido-3 O-[(R -1-carboxyethyl]-2-deoxy- d-glucose; heR -1-carboxyethyl]-2-deoxy-pR -1-carboxyethyl]-2-deoxy-omatogR -1-carboxyethyl]-2-deoxy-/MS) method, using C-labeled cyanobacteR -1-carboxyethyl]-2-deoxy- (Sebastian et al. 2004).
Therefore the relevant measure that we quantify is the rate of deformation, which is the rate of pure shear along the PD axis.
The third measure
As another measure of purity we quantified fragments of synaptic membrane bilayers under EM.
As a measure of healthspan, we quantified the number of body bends throughout adulthood.
Using a third assay to measure apoptosis induction, we quantified annexin-V staining by flow cytometry.
Although we did not measure enzyme activity, we quantified the abundance of mRNA transcripts coding for several detoxifying enzymes in cells treated with insulin or palmitate.
To measure oxidative stress, we quantified lipid peroxidation in serum and kidney tissue adjacent to the graft using the TBARS method [ 28].
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