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Different mean wave speeds and RMS errors were calculated for the seasons 2010 2011 and 2011 2012.
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The mean wave speed v is calculated as follows.
Mean shear wave speeds were >200% greater in sclerotic lesions than in contralateral healthy skin in patients with graft-versus-host disease (GVHD) (p < 0.01), and 25% greater in patients with morphea.
Whereas mean acoustic wave speed in TGF-β and GDF6-treated BM-MSC constructs was growth factor independent, acoustic-wave speed was significantly higher in TGF-β-treated AD-MSC seeded constructs compared with their GDF6-treated counterparts.
These MSC source and growth factor-related differences in fibrillar collagen content were, in turn, correlated with the mean acoustic wave speed (and hence stiffness) of the tissue constructs.
However, the mean wet-snow wave speeds for both years will lead to errors of 12.5 cm (2010 2011) and 23.3 cm (2011 2012).
Shorter AGWs with smaller horizontal wave speeds produce smaller mean heating and wave-induced mean velocities in the upper atmosphere at fixed amplitudes and periods of the surface wave excitation.
It remains debatable, whether a mean wet-snow wave speed is valid for other test sites and whether RMS errors stay comparably low over a full wet-snow season.
This means that the wave speed through the dry skull and polyurethane of the FE model is comparable to that in the experiment.
In all four case study sites, maximum current speeds increase with increasing magnitude of the earthquake source, increasing mean wave period or increasing scenario wave heights.
However the shape of the primary wave ring can differ indicating a seasonal variation in the azimuthal wave speeds, which is likely due to a seasonal variation of the mean flow and the thermal structure of the lower troposphere.
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