Sentence examples for mean pulse pressure from inspiring English sources

Mean pulse pressure was higher in the ITD group versus the fluid resuscitation group (P < 0.05).

SBP values obtained from 1997 to 1998 to time of neuroimaging were used to compute mean; pulse pressure; coefficient of variation; and peak, load, and group-based trajectories.

For each of the two lead biomarkers (blood lead and tibia lead), we used multiple linear regression to compare the mean pulse pressure across quintiles of the lead biomarker.

Men with tibia lead level above the median (19.0 μg/gm) had, on average, a mean pulse pressure that was 4.2 mmHg greater [95% confidence interval (CI), 1.9 6.5] than men with tibia lead levels below the median.

The data suggest that there may be a threshold to the effect of lead exposure on pulse pressure, because only the top two quintiles of tibia lead were associated with a mean pulse pressure greater than that of the first quintile.

Mean daytime, nighttime, and 24-h blood pressure as well as mean 24-h pulse pressure, mean arterial pressure, and mean 24-h heart rate decreased significantly over the course of the 3 months of treatment (p < 0.001; Fig.  2a, b).

As shown in Table 2, although HFOV did not significantly decrease arterial pressure (systolic, diastolic, mean and pulse pressure), it significantly decreased CI by 13 ± 11%% from 3.7 ± 1.1 L min−1 m−2 at baseline and LVOT VTI by 13 ± 12%% from 17 ± 5 cm at baseline.

Hemorrhage (median blood volume loss, 33 ± 7 mL, ≈15 mL/kg) induced a significant decrease of AoF (P < 0.05), meanwhile systemic arterial pressure (mean and pulse pressure), C, TPR and Eadyn did not show a significant change.

Although HFOV did not significantly decrease the arterial pressure (systolic, diastolic, mean and pulse pressure), it significantly decreased the cardiac index (CI) by 13 ± 18 % and significantly increased the RVEDA/LVEDA ratio by 14±11%14±11%

The existence of a transmantle gradient (either in mean or pulse pressure) has been hypothesized as one possible explanation for ventricular dilation in HC [ 107, 108], although a recent study by Eide and Saehle in NPH patients showed no evidence of pulsatile trans- or intra-mantle pressure gradients [ 2].

As shown in Table  2, although HFOV did not significantly decrease arterial pressure (systolic, diastolic, mean and pulse pressure), it significantly decreased CI by 13 ± 11%% from 3.7 ± 1.1 L min−1 m−2 at baseline and LVOT VTI by 13 ± 12%% from 17 ± 5 cm at baseline.