Exact(2)
Comparison of the mean peak currents and mean peak power densities revealed that MFCs run on cellulose were electrically outperformed by those run on starch but not those run on chitin.
Mean peak currents are shown.
Similar(58)
Figure 3A, which presents mean peak current voltage-relationships for WT (open triangles) and F1473C (filled squares) channels, indicates no mutation-induced change in mean current density.
Comparing the mean peak current amplitude across early postnatal weeks demonstrates the developmental upregulation of nicotinic excitation in male and female layer VI neurons as illustrated in Figure 1B.
(G ) Normalized mean peak current as a function of prepulse duration for cells transfected with Piezo1.
Fig. 11B presents mean peak current amplitudes recorded following injection of various combinations of scn8aa ± β1 subunits.
(F ) Mean peak current as a function of prepulse duration for cells transfected with pcDNA or Piezo1 (N = 9 and N = 11, respectively).
The current voltage (I V) analysis revealed that the mean peak current amplitude was significantly higher in CCL2-treated neurons than in control neurons (64.28±3.12 pA/pF versus 46.53±4.33 pA/pF, P<0.05).
Mean peak current upon stepping to +50 mV was reduced from 1,625 ± 238 pA to 776 ± 127 pA in the presence of 1 mM TEA, a mean reduction of 52% (n = 9; P < 0.001).
(B ) Mean peak current upon release of a 5 s positive pressure pulse (0 to +10 mmHg) for cells transfected with empty vector (pcDNA; N = 9 cells) and with mouse Piezo1 (N = 14 cells).
However, the mean peak current amplitude of Nav1.8 in DRG neurons was significantly reduced after being treated with CCL2+CCR2 inhibitor INCB3344 (10 nM) compared with the control and CCL2-treated neurons (P<0.05), whereas no significant change in Nav1.8 current density was observed when INCB3344 (10 nM) was applied alone.
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