Sentence examples for mean microvessel density from inspiring English sources

For a mean microvessel density of 1 miCarpenter/mm2 (Carpetter et al., 2011), we find good agreement of the fractional probability of at least one hot spot with the observed invasive tumor occurrence (Morris & Kwong, 2004) in different regions of the breast.

Defining a hot spot to be a circle of radius 200 μm with at least 5 microvessels, and using a previously measured mean microvessel density of 1 microvessel/mm2, we find good agreement of the fractional probability of at least one hot spot in different regions of the breast with the observed invasive tumor occurrence.

Three high magnification fields were counted for each tumour section and the mean microvessel density value was recorded for each.

Mean microvessel density (MVD per mm) and pericyte coverage index (PCI) were calculated after analysing at least 20 random fields per slide (Table 3).

Patients with increased mean microvessel density (MVDmean > 68 mm2) had a significantly shorter 5-year survival compared with the rest (57% vs 90%, P = 0.004).

Mean microvessel density in PET negative metastases was comparable with that found in PET positive nodules (12.9±3.9 vs 11.3±3.5 no significant difference).

To test our model, we used the two published 2D mean microvessel densities of 1 (Carpenter et al., 2011) and 61 (El-Gohary et al., 2009) microvessels/mm2009

The mean visual microvessel density was calculated as the average of four counts (two of the authors: two microscopic fields).

As shown in Table 3-wrap>, mean intratumoural microvessel density was 11.9±3.7 (median 12), IMD was independent from nodule size (P-value=0.89) being significantly higher in sarcomas than in colon cancer (mean microvascular density: 16.6±1.7 vs 10.2±3.2, respectively; P<0.0001).

There was no correlation between mean ECP% and microvessel density (both Chalkley and Weidner method), mean percentage of immature vessels or stromal fibrin deposition.

In fact, the mean hot spot microvessel density did not differ significantly among individual D-12 tumours, perhaps because they were derived from the same cell line and thus, in contrast to the clinical tumours, had the same genetic background.