In contrast, recently constructed housing units were associated with decreased mean lead levels.
For HFE genotype, results were nonsignificant, but it may be noted that in both men and women the minor allele homozygotes (YY or DD) had the lowest mean lead levels.
To achieve high BLLs for the experiments on the mechanism underlying lead-associated nephrotoxicity, we treated rats with 1,000 ppm Pb2+ in drinking water; in lead-treated rats, the mean BLL was 73.5 ± 17.4 μg/dL and the mean lead levels in kidney tissue was 26.67 ± 3.67 μg/g.
Finally, geometric mean lead levels during all trimesters of pregnancy in a random sub-sample (n = 105) were between 5.5 ± 1.8 μg/dL at first trimester and 5.6 ± 1.9 μg/dL at third trimester, with a maximum of 27 μg/dL.
For example, in one study of approximately 1,000 subjects 50 70 years of age with mainly environmental lead exposure, the mean blood lead level was approximately 4 μg/dL and the mean tibia lead level was approximately 19 μg/g, but the Pearson's r correlation between the two was only 0.12 at a time of low ongoing environmental lead exposure (Martin et al. 2006; Schafer et al. 2005).
In summary, in rats nonpoisoned with lead, the mean blood lead level was lower than 100 μg/L, which means it corresponded to the environmental exposure to this metal [ 7, 43– 43].
Mean blood lead level was significantly greater among the urban subgroup, as was the mean number of carious tooth surfaces.
The mean age of children undergoing the challenge test was 38.2 months, and the mean blood lead level was 1.83 micromol/L (38 microg/dL).
The mean age of the population was 54.7 years (SD = 9.8), with a mean blood lead level of 9.2 microg/dL (SD = 4.7/dL) and a range from 2.1 to 32.1 microg/dL.
