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Lower mean lactate levels have been demonstrated in survivors than in nonsurvivors among dogs with SIRS [ 37].
Five recent outcome trials comparing various approaches in diagnosing acid base disorders had relatively low mean lactate levels: 2.7 mEq/l in survivors [ 40]; 1.88 mEq/l [ 24]; 1.0 mEq/l [ 30]; 2.3 mEq/l in survivors [ 20]; and 3.1 mEq/l [ 15].
All patients showed rewarmed extremities, decrease of mean lactate levels (2.44 ± 1.33 mmol/l (1.10 5.4)).
These findings are different from a previous study of mild hypothermia therapy in patients with traumatic brain injury, in which the hypothermia group showed lower mean lactate levels and improved neurologic outcome [ 30].
Mean lactate levels in cerebrospinal fluid rose consistently from the beginning of the intervention steadily until after surgery (T1 = 1.83 mmol/l, T2 = 2.10 mmol/l, T3 = 2.72 mmol/l, T4 = 3.70 mmol/l, T5 = 4.31 mmol/l).
Mean lactate levels were surprisingly low in both survivors (2.7 mEq/l) and nonsurvivors (4.0 mEq/l), whereas SIG levels were elevated in both (9.7 mEq/l and 15.9 mEq/l, respectively).
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The mean lactate level was 39.5 mg/dL on those alive and 97.6 mg/dL on the dead.
The mean lactate level in patients on stavudine was 1980 μmol/l (S.D. ±1197) versus 1051 μmol/l (S.D. ±395, P=0.01) in patients on zidovudine.
The mean lactate level was 9.6 mmol/L and mean CPR duration was 28.8 min.
The mean lactate level was 9.6 mmol/L and the mean CPR duration was 28.8 minutes.
The mean lactate level was 3.3 mmol/L in both placebo and treatment groups.
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