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We found a significant increase both in single producing (p = 0.0136, mean increased from 0.061 to 0.079%), double producing (p < 0.0001, mean increased from 0.013 to 0.036%) and triple producing (p = 0.0004, mean increased from 0.002 to 0.018%) T cells after stimulation with the HAC1 protein (Fig. 6B).
Crude comparisons of the effects within groups showed that VO2peak mean increased from 1.81 at baseline to 1.87 l O2/min at follow-up (ie, an increase by 3.3%) in the intervention group whereas the change in the control group was 1.69 vs 1.73 l O2/min (ie, an increase by 2.3%).
The mean increased from 22.10 to 48.62 mg/g with an increase in dye concentration.
Among those who had at least 1 publication, the mean increased from 1.3 to 2.2 (range 1 15).
The IG scored higher at follow-up than at baseline (P < 0.001); the mean increased from 6.4 to 9.0 out of a maximum 20 points.
For PFNA, however, the percentage of serum samples with detectable levels increased, and the geometric mean increased from 0.5 ng/mL to 1.0 ng/mL, between the NHANES waves.
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Control means increased from 0.316 ± 0.01 (8) (mean ± S.D. (n)) at baseline to 0.323 ± 0.008 (10) but showed no significant difference over 0 – 6 weeks.
45 The corresponding arithmetic means increased from 0.9 minutes at baseline to 3.1, 3.6, and 1.9 minutes, respectively (both P < 0.001 vs placebo, Table 2).
Treatment with valsartan and placebo was associated with least-squares mean increases from baseline to week 14 in distance on the 6 min walk test (15.6 and 12.7 m, respectively).
Least-squares mean increases from baseline to endpoint in exercise time, the primary efficacy variable, were observed in both study groups (Table 3 ), with the improvement on placebo (1.24 min) being slightly better than on valsartan (0.96 min).
Log SP (daily mean) increasing from 15 micrograms/m3 to 331 micrograms/m3 (5% quantile to 95% quantile) was associated with a 22% increase in mortality.
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