Sentence examples for mean diffusivity found from inspiring English sources
In conclusion, our results suggest that the increase in mean diffusivity found in different brain regions reflect the process of cytotoxic edema in vivo.
Increases were also found in axial diffusivity as well as in mean diffusivity (see Fig. S2 and Table S2).
We repeated the final model substituting the median fractional anisotropy value with median mean diffusivity, and found consistent results [χ(2) = 3.6, P = 0.16; CFI = 0.99; RMSEA = 0.08, 90% CI = 0.0 0.22].
An increase in mean diffusivity was again found in similar regions as fractional anisotropy reduction, although not all regions with fractional anisotropy reduction showed an increase of mean diffusivity (Fig. 3).
A positive correlation between action binding and mean diffusivity in patients was found in several white matter tracts (P < 0.05, FWE corrected; tract listed in Supplementary Table 3).
The highest variability in mean diffusivity (MD) values was found for the pubovisceral muscle (1.49 ± 0.47 × 10−³ mm²/s) and might be attributed to the complex organisation of this muscle.
One voxel-wise DTI study [ 40] found that mean diffusivity (MD) and radial diffusivity (RD) representing the diffusivity in directions perpendicular to fiber orientations increased significantly in most brain regions in ESRD patients.
We found differences in mean diffusivity (MD) between EM and CM patients in the left superior longitudinal fasciculus temporal (Table 1).
This study also included diffusion imaging and found associated changes in mean diffusivity in the caudate of presymptomatic mutation carriers.
We tested the diagnostic value of diffusion tensor-derived axial diffusivity (AD), mean diffusivity (MD) (=apparent diffusion coefficient) and radial diffusivity (RD) maps in comparison to T1w and T2w sequences.
To conclude, we found widespread fractional anisotropy, mean diffusivity and axial diffusivity differences between patients with traumatic brain injury and healthy controls using TBSS.
