Sentence examples for mean cell death from inspiring English sources

In CA1 the mean cell death at all time points was slightly higher in BK−/− than in WT cultures even without IVI (F 1,46) = 2.65 n.s).s

The mean cell death rate was approximately equal to 1% of the average division rate.

Untreated controls retained a mean cell death percentage of less than 1% at 24 hours, 48 hours and five days.

Similarly, the silencing of Puma or Noxa did not prevent the Len/VD3-induced cell death: the mean cell death was 21±2%, 21±1% and 22±2% in siCt, si NOXA and si PUMA cells, respectively.

SNP effectively induced cell death (p<0.001, all time points) with mean cell death of 92.95 ± 2.29% at 24 hours, 99.6 ± 0.17% at 48 hours and 100 ± 0.00% at five days.

Similarly, the transfection with siBAX reduced by 68±4% the Len/VD3-induced cell death (P=0.01), but it did not significantly induced cell death in untreated cells (P=0.25): the mean cell death was 11±2% and 28±6% in untreated and Len/VD3-treated siCt cells, respectively, versus 12±3% and 18±5% in untreated and Len/VD3-treated si BAX cells, respectively.

The speed is important because it means that cell death doesn't occur and the tissue being printed remains viable, while the ability to print within structures means that Prellis' technology can generate the internal scaffolding to support and sustain the organic material that surrounds it, according to the company.

The basic findings confirm the above HeLa cell data, but the faster utilisation of arginine meant that cell death was more rapid in its absence, whether by AFM or ETM treatment.

Data were displayed as mean % cell death to normalise differences in absolute cell number between experiments.

Our data suggest that cells already have a mechanism to inactivate Chk1, therefore Chk1 inhibitors might accelerate a process that occurs naturally in cells rather than induce a new means of cell death.

DNA laddering was observed in tumor cells of mice treated with pcDNA 3.1-ANT1 (Fig 4B), which means that apoptotic cell death rather than cell cycle arrest might be involved in the suppression of tumor growth by pcDNA 3.1-ANT1 treatment.