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Yet, coronary atherosclerosis has been suggested to be an inflammatory disease in which chronic dental infections may trigger pathogenic mechanisms in the walls of arteries.
Intercellular spreading of DPR proteins may trigger pathogenic mechanisms leading to TDP-43 phosphorylation or seed TDP-43 aggregation in a non-cell autonomous manner.
Bacterial toxins may trigger pathogenic events through the over-production of cytokines and chemokines, leading to the alteration of endothelial function and capillary leakage [ 25].
These observations may have relevance in cases that present with flaccid paralysis because bacterial infections, co-infections or post-infection immune responses may trigger pathogenic processes that may present as poliomyelitis-like syndromes and may mimic AFM.
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The sensitization stage may trigger additional pathogenic mechanisms contributing to the drug-induced neural changes conducive to TD, such as the sensitivity of dopamine D1 and serotonin 5-HT2C receptors [ 38], and/or lessening the endogenous adaptive response mounted to fend off TD.
The changes may trigger some pushback.
Alternatively, insomnia may trigger TTH [32].
Such events may trigger new PuLSaR recommendations.
Everything may trigger you.
Certain pesticides may trigger autism.
The Hesson name may trigger memories.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com