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Common ingredient in packaged food may trigger inflammatory disease.
Taken together, our data suggest that the abnormal SMCs may trigger inflammatory infiltrate while normal SMCs should have some mechanisms to suppress or bypass the pro-inflammatory cytokine signals.
Data in animals suggest that oral iron sulfate may trigger inflammatory processes associated with progression with Crohn's disease-like ileitis (34).
It is most likely that DAMPs released from the necrotic cells and tissues resulting from their exposure to toxic molecules, such as ROS, may trigger inflammatory response.
Increased plasma IgE may trigger inflammatory cytokine expression from T cells, MCs, macrophages, and other inflammatory cells, thereby promoting AAA pathogenesis.
Mg deficiency may be exacerbated by low Mg with high or low Ca intakes [ 13, 28], which may trigger inflammatory stress, contributing to the development of CVD [ 13].
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Tissue creep may also trigger inflammatory processes and enhance muscle spasm [ 30].
Moreover, HMGB1 may trigger an inflammatory response and contribute to the pathological progression of infectious and inflammatory disorders, such as enhancing the production of TNF-α, IL-1β and nitric oxide [ 9, 10].
Infection often results in tissue damage, which may trigger an inflammatory response.
Mechanical ventilation may trigger an inflammatory response, comprising alveolar macrophage activation and recruitment, which may be specifically, repeatedly and spatially assessed by functional imaging techniques such as Positron Emission Tomography combined with Computerized Tomography (PET/CT) [2].
These results indicate that accumulation of VLC fatty acid contents in membrane domains associated with signal transduction pathways may trigger the inflammatory process through activation of resident glial cells (microglia and astrocytes) resulting in loss of myelin and oligodendrocytes.
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