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We may therefore speculate that chromosome 19 pericentromeric loci comprise specific recombination-prone genomic structures leading to chromosomal instability by recombination-driven rearrangements.
One may therefore speculate that germ cells could be specifically susceptible to genotoxic chemicals which cause some types of bulky DNA adducts.
We may therefore speculate that infection of farms across the M6 was exclusively via road.
One may therefore speculate that 20S proteasomal degradation is yet another process of metabolic sensing by which UCP2 may oppose p53 responses and support cancer cell survival.
We may therefore speculate that combination of these frequent mutations will worsen the phenotype and could be a common but under-diagnosed phenomenon.
We may therefore speculate that the benefits of obtaining a higher PLAB part 1 score, which is knowledge based, may generally only be realised at ARCP in the presence of appropriate clinical exposure and training.
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We therefore speculate that NLK may also play a role in protein clearance pathways, and that this, in turn, may contribute to the ability of NLK to regulate mutant protein aggregation and toxicity in varying disease cases.
We therefore speculate that TUDUSP7 may function as a binding site for some other USP7-binding proteins or substrates.
We can therefore speculate that this may also be the case for the APP and tau IRESes, and this is currently under investigation.
We therefore speculate that metformin may raise ROS levels to activate intestinal SKN-1, where it functions to defend against oxidative stress and promote longevity.
We therefore speculate that tumorigenesis may be associated with long-term exposure of petroleum products.
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