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Alterations of afferent sensory information, potentially caused by mechanoreceptor damage, may subsequently contribute to disturbances of sensorimotor control [1, 25, 59].
The accumulation of these inflammatory cells may subsequently contribute to the development of airway hyperresponsiveness, local inflammation, and tissue injury through the release of granular enzymes and other cytokines.
These cells may be attracted by the tumors' hypoxic areas and necrosis, and may subsequently contribute to the inflammatory signature (enriched on the negative side of the loading plot) within the cluster of hypoxia-inducible genes.
Such research may subsequently contribute to developing effective programmes to address the needs of urban refugees.
Autophagy induction may subsequently contribute to overcoming nutrient deprivation by future recycling the existing proteins.
Moreover, an imbalance between clot formation and hyperfibrinolysis as a result of systemic hypoperfusion and anticoagulation may subsequently contribute to consumption coagulopathy and bleeding disorders [ 5, 7].
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Pre-existing beliefs that the back needs protection to prevent injury may be reinforced by experiencing back pain following a trivial injury, and may also subsequently contribute to the development of avoidant behaviour.
An ancient "shopping for plastidial eukaryotes" [ 24] in the heterotrophic ancestor of the heterokonts/chromalveolates could explain the origin of the metabolic versatility that may have subsequently contributed to the ecological success of this group of organisms irrespective of whether photosynthesis was retained or not [ 77, 78].
This may subsequently lead to cell death.
In contrast, we argue that mirror neurons may develop through associative learning and subsequently contribute to action understanding.
However, in the presence of 4HPR, the decrease in ubH2A was significantly greater than in the vector control cells; this suggests that ectopic Uba80 or Uba52 expression may sensitize the cells to apoptosis and subsequently contribute to H2A deubiquitylation.
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