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It is possible that IL-6 expression or hydro-dynamic injection may stimulate expression of TNF-α and MCP-1, which was not sufficient to induce liver injury.
Therefore, it is tempting to speculate that high levels of circulating IFNγ in AKR/J mice may stimulate expression of Adcy7 in immune cells, with concomitant effects on intracellular cAMP levels and intensity of inflammatory response in these mice.
RLX is generally an ER antagonist in breast tissue; however, RLX may stimulate expression of certain ER genes.
Several reports have suggested that elevated expression of COX-2 may stimulate expression of MDR1/P-gp.
Although these sequences have not yet been identified, an algorithm to analyze introns that may stimulate expression in plants has been developed.
We hypothesized that, using long-term dietary intervention studies, dietary fatty acid composition may modulate gene expression and lipid metabolism in the intestine, and that especially EPA and DHA may stimulate expression of genes involved in lipid catabolism.
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LPS may also stimulate expression of stress-induced proteins, such as MIC-A and MIC-B (ligands for natural killer cell activating receptors) and T cell co-stimulatory molecules (B7 family proteins).
Previously published data indicate that the basal cell specific transcription factor ΔNp63 may either stimulate expression of Notch receptors (Laurikkala et al., 2006) or inhibit their expression (Nguyen et al., 2006; Okuyama et al., 2007; Yugawa et al., 2010; Romano et al., 2012), depending on the tissue examined and the developmental stage.
Given that E2 acts as a potent stimulator of IGF1 signaling in the endometrium (Ivanga et al, 2007) and that H19 stimulates Igf1r expression (Fig 2; Appendix Fig S2), we hypothesized that E2 may stimulate H19 expression which in turn upregulates Igf1r expression, thereby promoting IGF1 signaling in the human endometrial cells.
Previous studies have indicated that hypoxia may stimulate gene expression through different pathways, including ERK, JNK, p38 MAPK and PI3K/Akt [ 30], and therefore hypoxia may increase the expression of myocardin through one of these pathways.
Furthermore, corticotomies may stimulate the expression of inflammatory markers and cytokines which leading to increased osteoclast activity [16 19].
More suggestions(15)
may modulate expression
may stimulate osteolysis
may stimulate dynein
may exhibit expression
may prevent expression
may affect expression
may stimulate muscle
may regain expression
may promote expression
may show expression
may find expression
may increase expression
may stimulate proliferation
may cause expression
may stimulate water
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