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PPARγ also may regulate neuronal responses to ischemia since conditional deletion of PPARγ in neurons increases their susceptibility to ischemia in vitro (Zhao et al., 2009).
Thus, LIS1-mNudE interactions may regulate neuronal migration through dynamic reorganization of the MTOC.
Downregulation was observed in cryptochrome circadian clock 2 (CRY2), which influences the circadian rhythm and in BCL2L2, which may regulate neuronal death and the survival of oligodendrocytes.
On one hand, DGKβ may regulate neuronal morphogenesis through PA production because it is also a lipid messenger.
These results suggest that RGS14 may regulate neuronal differentiation by the selective organization of a Ras·GTP-dependent Raf, MEK, and ERK signal transduction complex in vivo.
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These results show that Znf179 may also regulate neuronal cell fate determination.
This literature is most prevalent in the brain where caspase-1 may directly regulate neuronal cell death in response to diverse insults.
Taken together, these results demonstrate a radial glial signaling mechanism that may regulate cortical neuronal production.
Alternatively, FruM may regulate dopamine neuronal activities.
The transcriptional control of neuropeptide expression is not the only way by which insulin and leptin may regulate the neuronal circuits controlling food intake.
Better understanding of how these distinct mechanisms are orchestrated to regulate neuronal activity may lead to the design of better treatment strategies for patients with epilepsy, as well as for other hormone-sensitive brain disorders.
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