Dopamine is further involved in memory processes by modulating neurons in the prefrontal cortex that are active during the delay interval between a stimulus presentation and a response [ 44], and may regulate working memory and attention [ 13, 45].
However, the C1858T PTPN22 mutation may also promote autoimmunity via other pathways that regulate memory B-cell formation [ 15] and dendritic cell (DC) responsiveness [ 16].
In the present study, Svr functioning in a restricted group of IPCs dilp2 neurons to regulate memory formation implies that the potential connection between Svr and insulin pathway may be involved in memory formation (Fig. 2D).
Breton, J. & Robertson, E. M. Flipping the switch: mechanisms that regulate memory consolidation.
Thus, the G protein-coupled mtCB1 receptors regulate memory processes via modulation of mitochondrial energy metabolism.
Breton, J. & Robertson, E.M. Flipping the switch: mechanisms that regulate memory consolidation.
DNA methylation and histone acetylation work in concert to regulate memory formation and synaptic plasticity.
In summary, MR and GR expression in the LA was examined because the LA is the site of fear memory formation [1], [3], [76] and activated mGR and mMR may regulate the acquisition of that memory [4], [7], [8], [16], [17].
It is known that, besides its effects on the components of feeding circuits, leptin also acts as a cognitive enhancer in the hippocampus and on excitatory synaptic strength, which may regulate the processes involved in learning and memory 17, 36.
In this way, α-synuclein may regulate the release of dopamine in controlling voluntary and involuntary movements, or might influence memory and cognitive function as shown in SNCA knockout mice [ 29].
