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This impairment may promote vulnerability, especially of striatal medium-sized spiny neurons, to the excitotoxic damage and their degeneration (e.g., [ 12, 13]).
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This may further promote vulnerability, especially of striatal medium-sized spiny neurons, to the excitotoxic damage [ 12].
This may partly reflect the multi-factorial elements that lead to FGR as well as subsequent environmental influences that promote vulnerability to polygenic disease, such as type-2 diabetes mellitus (T2DM).
It has been hypothesized that a persistent lack of cortisol availability in traumatized or chronically stressed individuals may promote an increased vulnerability for the development of stress-related bodily disorders [ 51].
It has been postulated that impairment of myocyte cell-to-cell adhesion may lead to tissue and organ vulnerability, which may promote myocyte death especially during mechanical stress, which occurs during competitive sports activity.
Intraplaque hemorrhage (IPH) may cause complications by promoting vulnerability, luminal occlusion or downstream emboli.
Dissolved organic matter-induced Mn oxide dissolution may promote repartitioning of DOM into the aqueous phase, increasing the vulnerability of DOM to microbial attack relative that sorbed to minerals surfaces.
As DYN expression is increased in the striatum following chronic drug use [ 347, 371- 373], the formation of μOR/κOR dimers may promote the engagement of very different signaling pathways in females, which could exacerbate or attenuate their vulnerability to addiction.
Finally, selective-use anticoagulation may promote patency.
Treatment with antibiotics may promote Proteus growth.
This may promote misdiagnosis of this injury.
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