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Correa et al [ 29] have clearly shown in a large trial conducted over six years that dietary antioxidant supplementation alone (vitamin C or beta-carotene), without antimicrobial therapy, can limit the progress of gastric cancer and actually may promote regression of precancerous states.
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Several mechanisms have been proposed by which angiotensin II inhibition may directly promote regression of renal injury.
These in vitro observations are supported by the Apcmin animal model [ 32] and sustained clinical trial in patients with H. pylori gastritis [ 30] suggesting that dietary antioxidants do not exacerbate precancerous states but may indeed promote regression.
The pathology suggests that tumour-infiltrating lymphocytes (TIL) and macrophages may promote CTVT regression [ 21, 22] and overall, the available data suggest that the immune system plays an important role in regression of CTVT.
Notably, these therapies may halt and even promote regression of renal injury in some patients.
The use of condoms helps promote regression of these changes.
The role of therapeutic vaccines against HPV is to promote regression of HPV-related lesions.
ApoE also has antioxidant properties [ 30] and can promote regression of atherosclerosis [ 31].
Disruption of the interaction of leukemic cells with their microenvironment may promote their apoptosis and leukemia regression.
Finally, selective-use anticoagulation may promote patency.
This may promote misdiagnosis of this injury.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com