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The storage moduli of the composites approached the lower range of storage modulus for cortical bone and may prevent stress shielding during bone regeneration.

The storage moduli of the nanocomposites at 37 °C approached the lower range of the storage modulus for cortical bone and this may prevent stress shielding during bone regeneration.

Among a general adult population, regular participation in PA may prevent stress by creating a better social network, higher stress tolerance, better self-esteem, and more active coping behaviors [ 21, 24].

In addition, the load is thought to be naturally transferred to the proximal femur, which may prevent stress shielding (Harty et al. 2005, Hayaishi et al. 2007, Little et al. 2007, Lian et al. 2008), thereby preserving bone stock postoperatively.

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We believe that this feature may prevent stress-shielding and load by-pass in a proximal distal direction, avoiding bone resorption of the proximal portion of the femur with subsequent secondary instability of the implant, even if the role of stress-shielding (and particularly its influence on clinical outcome) is still not clearly demonstrated [20, 34, 35, 36, 37, 38, 39, 40, 41, 42].

Lack of eif3e may prevent stress-induced synthesis of its critical translational targets, thus leading to the known stress sensitivity of the mutant.

In other words, by providing employees with necessary, matching job resources and recovery opportunities for coping with job demands, hospitals may prevent unnecessary stress and strain, and improve worker well-being and job performance.

We proposed that T17M rhodopsin may be misfolded and eliminated via ERAD, and chemical chaperone 4-phenylbutyrate may prevent ER stress induced by rhodopsin T17M.

An important point in this model is that a high degree of job autonomy and influence at work may prevent work stress, even when job demands are high.

The evidence for a period out of service post-CI (downtime) is sparse; however it may prevent posttraumatic stress disorder (PTSD) and burnout symptoms.

There are two main mechanisms by which organisms may prevent oxidative stress: (i) by increasing antioxidant defences in tissues, and/or (ii) by reducing mitochondrial ROS production.

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