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Patients with severe, acute complications may present typical symptoms such as abrupt headache, nausea, vomiting, consciousness alterations, myoclonus, tremor, and focal or generalized seizures.
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Patients may present with typical but non-specific symptoms of hypercalcaemia including malaise, depression, renal stones and abdominal pain or incidentally identified following identification of a raised serum calcium on blood biochemistry.
In this section, we present typical applications of the PRROC R-package.
Unfortunately, delays to or missed diagnosis are common especially related to patients with mild form since they may present without a typical coarse facies and with less pronounced skeletal disease.
Unique among atypical AD variants, temporal variant AD is a late-onset AD syndrome, and may present even later than typical AD.
Although cases with these typical signs may present little difficulty for a diagnosis of subacute thyroiditis, this disorder does not always present in a classic fashion and may lead to difficulties during diagnosis [ 5, 6].
These symptoms appear to occur in a typical pattern, but not all patients may present with or be able to account for a similar timeline.
Clinically, these congenital anomalies may present as cysts, sinus tracts, fistulae or cartilaginous remnants with typical clinical and radiological findings.
Small herds may present a problem in surveillance for infectious animal diseases because typical levels of a within-herd design prevalence are not directly applicable.
They may present as cysts, sinus tracts, fistulae or cartilaginous remnants and present with typical clinical and radiological patterns dependent on which arch is involved.
A small subset of β-catenin-activated HCAs may present with diffuse cytoplasmic expression of GS, but the map-like pattern typical of FNH is not observed.
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