One may predict that virus-induced host signaling would by applicable as novel medical modalities to a large spectrum of diseases.
In contrast, in terms of the hosts, we may predict faster change within the unselected (U) snails than in their previously resistant-selected (R) counterparts.
Nevertheless, findings from a longitudinal study of Southeast Asian refugees resettled in Canada indicate that persistent socio-economic hardship, particularly unemployment and poor host language proficiency, may predict depression even 10 years after resettlement [ 11].
For these reasons, further studies on host genetic factors that may predict the treatment outcome of combinational therapies and progression of liver disease are required.
These findings suggest that the immune status of the host at the time of aphaeresis may predict survival after ASCT.
Alternatively, as schistosomes have been demonstrated to display potential fitness compensatory responses through differential between-host-species transmission and establishment dynamics [ 23, 25], one may predict any observed changes in parasite infectivity, or virulence, to the co-selected intermediate host here may also influence these traits at the definitive host stage.
Thus, we may predict that deletion of RAGE or RAGE blockade suppresses interferon-γ-propagated macrophage activation and the hyperinflammation response that injures the host.
Economists may predict dire consequences.
These astrologists may predict the answer.
Besides host factors, the propensity of breast cancer to give rise to distant metastases depends on the molecular type of breast cancer, and the gene expression profile of the primary tumor may predict the risk of distant recurrence [ 5, 6].
